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PangenomeCOLN315NCTC8325NewmanUSA300_FPR375704-0298108BA0217611819-97685071193ECT-R 2ED133ED98HO 5096 0412JH1JH9JKD6008JKD6159JSNZLGA251M013MRSA252MSHR1132MSSA476MW2Mu3Mu50RF122ST398T0131TCH60TW20USA300_TCH1516VC40

Summary[edit | edit source]

  • pan ID?: SAUPAN005129000
  • symbol?:
  • synonym:
  • description?: phage protein
  • strand?: -
  • coordinates?: 5265979..5266522
  • synteny block?: BlockID0039810
  • occurrence?: in 18% of 34 strains

tmcT : prophage type I addiction module toxin TmcT [1]

Staphylococcal toxin-antitoxin systems provide a mechanism to rapidly alter growth rates under changing environmental stresses. TmcT is a DUF2829 (SA1803) family toxin thought to inhibit membrane translocation systems including type VII secretion systems, NorA pumps and monovalent cation antiporters resulting in bacteriostasis and persistence. It is sequestered by the RNA antitoxin TmcA. Although not often identified on initial annotations, TmcT is actually present in several prophage genomes including USA300_FPR3757. The TmcTA module appears to have integrated into a recombination hotspot distal to the prophage φ12-like mor regulator, replacing the n32 core sequence in: ACAAACA - n32 - ACAAACA

Orthologs[edit | edit source]

    COL:
    N315:
    NCTC8325:
    Newman:
    USA300_FPR3757:
    04-02981:
    SA2981_1957
    08BA02176:
    11819-97:
    6850:
    71193:
    ST398NM01_2956
    ECT-R 2:
    ED133:
    ED98:
    HO 5096 0412:
    JH1:
    JH9:
    JKD6008:
    JKD6159:
    JSNZ:
    LGA251:
    M013:
    MRSA252:
    SAR2098
    MSHR1132:
    MSSA476:
    Mu3:
    Mu50:
    MW2:
    RF122:
    ST398:
    T0131:
    TCH60:
    TW20:
    SATW20_19930
    USA300_TCH1516:
    USA300HOU_2002
    VC40:

Genome Viewer[edit | edit source]

Alignments[edit | edit source]

  • alignment of orthologues:

  1. Gul Habib, Jiade Zhu, Baolin Sun
    A novel type I toxin-antitoxin system modulates persister cell formation in Staphylococcus aureus.
    Int J Med Microbiol: 2020, 310(2);151400
    [PubMed:32001143] [WorldCat.org] [DOI] (I p)